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• MY Research Focused on Bronchopulmonary Dysplasia, A Condition of the Neonate Associated with Preterm Birth, Inflammation, and Hyperoxia. the Lung Develops at Low Oxygen Tension, and Aberrant Transition to Increased O2 Tension or Perinatal Inflammation Inhibits Alveolarization. i Focused on the Innate Immune Response Following Hyperoxia Exposure in Fibroblasts, A Critical Cell Type in Alveolarization . MY Data Showed an Increase in Tlr2 and Cytokine Expression Following Hyperoxia Exposure in Fetal Lung Fibroblasts, Which was Potentiated by Treatment with A Tlr2 Agonist in Hyperoxic Cells. This Suggests That Fibroblasts may Contribute to the Early Pro-Inflammatory State in the Hyperoxia-Exposed Preterm Neonate, and That Tlr Targeted Therapies may Promote Distal Lung Maturation. Publications: Gebb, S.A., Decoux, A., Waggoner, A., Wilson, G.L., Gillespie, M.N. Mitochondrial Dna Damage Mediates Hyperoxic Dysmorphogenesis in Rat Fetal Lung Explants. Neonatology. 103(2):91-97, 2013.
• Activities and Societies: Beta Beta Beta Biological Honor Societyas an Undergraduate, i Worked in A Lab That Focused on Recrudescence of West Nile Virus (Wnv) in A Migratory Species of Birds, the Gray Catbird. MY Project Focused on Changes in the Type and Number of White Blood Cells (Wbcs) in Birds Infected with Wnv. Further, A Correlation Between Testosterone and Immune Function had Been Reported, and MY Research Investigated Changes in Circulating Wbcs in Response to Increased Testosterone Levels.